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折扣/货期| 货号：SMC-405D-FITC | 规格：100 µg | 目录价：￥4800.6 |
产品详情
* 以下信息仅供参考，详情请以原厂网站为准
产品名称:
Cav3.1 Antibody: FITC
别名:
CACNA1G Antibody, CaV T1 Antibody, Cav3 1 Antibody, cav3 1c Antibody, KIAA1123 Antibody, MGC117234 Antibody, NBR13 Antibody, voltage gated calcium channel cav3.1 Antibody, calcium channel voltage dependent alpha 1G subunit Antibody, calcium channel voltage dependent T type alpha 1G subunit Antibody, calcium channel voltage dependent T type alpha1G subunit Antibody, voltage dependent calcium channel alpha 1G subunit isoform 11 Antibody, voltage dependent T type calcium channel subunit alpha 1G Antibody, Voltage gated calcium channel subunit alpha Cav3 1 Antibody
产品描述:
Mouse Anti-Mouse Cav3.1 Monoclonal IgG1
反应种属:
Human | Mouse | Rat
宿主来源:
Mouse
实验应用:
WB | IHC | ICC/IF
靶标/特异性:
Detects ~<200kDa. Does not cross-react with Cav3.2.
同种型:
IgG1
推荐稀释度:
WB (1:1000), ICC/IF (1:100);optimal dilutions for assays should be determined by the user.
免疫原:
Fusion protein amino acids 2052-2172 (cytoplasmic C-terminus) of mouse Cav3.1
免疫原种属:
Mouse
克隆性:
Monoclonal
克隆号:
N178A/9 (Formerly sold as S178A-9)
纯化方式:
Protein G Purified
偶联:
FITC
产品浓度:
1 mg/ml
保存温度:
Conjugated antibodies should be stored according to the product label
运输温度:
Blue Ice or 4ºC
存储溶液:
PBS pH7.4, 50% glycerol, 0.09% sodium azide *Storage buffer may change when conjugated
产地:
加拿大
功能与背景:
Calcium channel CaV3.1 (a1G) is a low-voltage-activated T-type calcium channel. Such T-type channels are expressed throughout the body. In the heart, they may be involved in pacemaker current. In neurons, these channels may play a secondary pacemaker role (1). With the ubiquitous expression, it is not surprising that alterations in channel function have been implicated in disease. Drugs that act to block T-type calcium channels are used as anti-hypertensives, antiepileptic's, and blocking of T-type calcium channels may be involved in the action of some anesthetics and antipsychotics as well (1). Much remains to be determined about the precise cellular localization, in vivo physiological roles, roles in disease states and possible routes to modulate their structure/function to ameliorate effects of disease.
Accession #:
NP_001106284.1
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